Skip to main content

A45 Coventry Road Safety Update

Further to our work and the petition that was raised on this matter last year, we recently met with a representative from Birmingham Community Safety Partnership and local Police Inspector James Gooderidge to discuss developments in how safety concerns on the A45 could be addressed.

We presented the views of our constituents who have conscientiously written letters and emails to our office and from this, the following points have now been forwarded to the BCC Highways Department to look into further;

  • The Tesco lane merger down towards to pedestrian crossing near Yew Tree Lane
  • Visibility for  drivers on the A45 into City (at the traffic light junction with the Bill & Bull onwards) there are 4 lanes of traffic to cross and only a single traffic light column on either side
  • Increased signage for pedestrians near to shops, parks etc to inform them where crossing are located. (i.e pedestrian crossing 50 meters this way → )
  • Continued monitoring of the data to assess how recent road alterations may have impacted upon both driver and pedestrian behaviour.
I will continue to post further developments in due course.

Comments

Popular posts from this blog

Its the long genes that stop working

People who read my blog will be aware that I have for some time argued that most (if not all) diseases of aging are caused by cells not being able to produce enough of the right proteins. What happens is that certain genes stop functioning because of a metabolic imbalance. I was, however, mystified as to why it was always particular genes that stopped working. Recently, however, there have been three papers produced: Aging is associated with a systemic length-associated transcriptome imbalance Age- or lifestyle-induced accumulation of genotoxicity is associated with a generalized shutdown of long gene transcription and Gene Size Matters: An Analysis of Gene Length in the Human Genome From these it is obvious to see that the genes that stop working are the longer ones. To me it is therefore obvious that if there is a shortage of nuclear Acetyl-CoA then it would mean that the probability of longer Genes being transcribed would be reduced to a greater extent than shorter ones.