Skip to main content

Results: Thursday 20th November 2008.

South Cambridgeshire DC, Teversham
LD Frances Amrani 269 (38.6; +2.9)
Con 237 (34.0; -30.3)
Ind 191 (27.4; +27.4)
Majority 32
Turnout 36.1%
LD gain from Con
Percentage change is since May 2007

Tower Hamlets LBC, Mile End East
Lab 1208 (47.3; +15.0)
Con 630 (24.7; +13.7)
Respect 604 (23.7; -0.3)
LD Jainal Choudhury 110 (4.3; -15.7)
Majority 578
Turnout 31.4%
Lab hold
Percentage change is since May 2006

Barnstaple TC, Forches and Whiddon Valley
LD Tony Clark 287 (51.3)
Con 164 (29.3)
Ind 97 (17.4)
Communist 11 (2.0)
Majority 123
Turnout 17.0%
LD hold

Comments

Popular posts from this blog

Its the long genes that stop working

People who read my blog will be aware that I have for some time argued that most (if not all) diseases of aging are caused by cells not being able to produce enough of the right proteins. What happens is that certain genes stop functioning because of a metabolic imbalance. I was, however, mystified as to why it was always particular genes that stopped working. Recently, however, there have been three papers produced: Aging is associated with a systemic length-associated transcriptome imbalance Age- or lifestyle-induced accumulation of genotoxicity is associated with a generalized shutdown of long gene transcription and Gene Size Matters: An Analysis of Gene Length in the Human Genome From these it is obvious to see that the genes that stop working are the longer ones. To me it is therefore obvious that if there is a shortage of nuclear Acetyl-CoA then it would mean that the probability of longer Genes being transcribed would be reduced to a greater extent than shorter ones.