Skip to main content

The Natives they are restless

Having treated Labour MPs as mere lobby fodder for many years it does appear that they are getting a bit restless at the moment.

Charles Clarke recognised that the government could actually lose parts of the terrorism bill in the House of Commons and so moved into consensus mode.

It is all rather odd actually as it is difficult to work out why the government are doing what they are doing with the Terror Acts.

Recently Terror Acts have been used to deal with heckling at the Labour Conference and a woman walking on a cycle path.

The Violette-szabo-museum could be guilty under the act for celebrating the sabotage that Violette Szabo committed in France as a British SOE agent during the second world war.

The eternal problem is that of defining "terrorism" - something which the government are not doing that well at.

The government's other substantial failing is in not understanding that they really need to act in a way that does not increase the amount of terrorism in the world. I am not sure that they understand that they have some material part of the responsibility for creating a world that is less peaceful.

Comments

Popular posts from this blog

Its the long genes that stop working

People who read my blog will be aware that I have for some time argued that most (if not all) diseases of aging are caused by cells not being able to produce enough of the right proteins. What happens is that certain genes stop functioning because of a metabolic imbalance. I was, however, mystified as to why it was always particular genes that stopped working. Recently, however, there have been three papers produced: Aging is associated with a systemic length-associated transcriptome imbalance Age- or lifestyle-induced accumulation of genotoxicity is associated with a generalized shutdown of long gene transcription and Gene Size Matters: An Analysis of Gene Length in the Human Genome From these it is obvious to see that the genes that stop working are the longer ones. To me it is therefore obvious that if there is a shortage of nuclear Acetyl-CoA then it would mean that the probability of longer Genes being transcribed would be reduced to a greater extent than shorter ones.